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As principal phase II detoxification enzymes, GSTs protect living cells by catalyzing the conjugation of glutathione (GSH) to a wide variety of electrophilic molecules of both endogenous and exogenous origin

Mechanistically, low CFTR expression impairs the expression of the chloride ion-sensitive with-no-lysine kinase 1 (WNK1) protein, thereby leading to activation of transient receptor potential cation vanilloid 4 (TRPV4)

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When a connective target suffers severe mechanical trauma, the resulting micro-tears rapidly drop local oxygen tension, creating an ischemic cellular environment

Abnormal gait, unexplained weakness or paralysis

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